Phenotype Switching: Tumor Cell Plasticity as a Resistance Mechanism and Target for Therapy

نویسندگان
چکیده

برای دانلود باید عضویت طلایی داشته باشید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Phenotype switching: tumor cell plasticity as a resistance mechanism and target for therapy.

Mutations in BRAF are present in the majority of patients with melanoma, rendering these tumors sensitive to targeted therapy with BRAF and MEK inhibitors. Unfortunately, resistance almost invariably develops. Recently, a phenomenon called "phenotype switching" has been identified as an escape route. By switching from a proliferative to an invasive state, melanoma cells can acquire resistance t...

متن کامل

P14: Mitochondria as a Target for Drug Therapy in Epilepsy

لطفاً به چکیده انگلیسی مراجعه شود.

متن کامل

Cell polarity as a tumor suppressive mechanism.

Cell polarity is a fundamental property of epithelial cells that confers spatial organization to signalling pathways regulating many aspects of cell physiology including survival, proliferation, and motility. Loss of epithelial organization and apical-basal polarity correlates with the acquisition of a malignant phenotype, and accumulating evidence suggests that loss of polarity signalling cont...

متن کامل

Cell Cycle Regulating Kinase Cdk4 as a Potential Target for Tumor Cell Treatment and Tumor Imaging

The cyclin-dependent kinase (Cdk)-cyclin D/retinoblastoma (pRb)/E2F cascade, which controls the G1/S transition of cell cycle, has been found to be altered in many neoplasias. Inhibition of this pathway by using, for example, selective Cdk4 inhibitors has been suggested to be a promising approach for cancer therapy. We hypothesized that appropriately radiolabeled Cdk4 inhibitors are suitable pr...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

ژورنال

عنوان ژورنال: Cancer Research

سال: 2014

ISSN: 0008-5472,1538-7445

DOI: 10.1158/0008-5472.can-14-1174